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Peru national football team
Men's association football team
This article is about the men's team. For the women's team, see Peru women's national football team.
Nickname(s) | La Bicolor (The Bicolour) La Blanquirroja (The White and Red) La Rojiblanca (The Red and White) Los Incas (The Incas) |
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Association | Peruvian Football Federation (FPF) |
Confederation | CONMEBOL (South America) |
Head coach | Óscar Ibáñez |
Captain | Pedro Gallese |
Most caps | Roberto Palacios (128) Yoshimar Yotún (128) |
Top scorer | Paolo Guerrero (39) |
Home stadium | Estadio Nacional |
FIFA code | PER |
Current | 40 (19 December 2024)[1] |
Highest | 10 (October 2017) |
Lowest | 91 (September 2009) |
Peru 0–4 Uruguay (Lima, Peru; 1 November 1927) | |
Peru 9–1 Ecuador (Bogotá, Colombia; 11 August 1938) | |
Peru 0–7 Brazil (Santa Cruz de la Sierra, Bolivia; 26 June 1997) | |
Appearances | 5 (first in 1930) |
Best result | Quarter-finals (1970, 1978) |
Appearances | 34 (first in 1927) |
Best result | Champions (1939, 1975) |
Appearances | 2 (first in 1952) |
Best result | Fourth place (1952, 1956) |
Appearances | 1 (first in 2000) |
Best result | Semi-finals (2000) |
Appearances | 11 (first in 1938) |
Best result | Champio • Oxidative Stress and Lipid Peroxidation Products in Cancer Progression and TherapyAbstractThe generation of reactive oxygen species (ROS) and an altered redox status are common biochemical aspects in cancer cells. ROS can react with the polyunsaturated fatty acids of lipid membranes and induce lipid peroxidation. The end products of lipid peroxidation, 4-hydroxynonenal (HNE), have been considered to be a second messenger of oxidative stress. Beyond ROS involvement in carcinogenesis, increased ROS level can inhibit tumor cell growth. Indeed, in tumors in advanced stages, a further increase of oxidative stress, such as that occurs when using several anticancer drugs and radiation therapy, can overcome the antioxidant defenses of cancer cells and drive them to apoptosis. High concentrations of HNE can also induce apoptosis in cancer cells. However, some cells escape the apoptosis induced by chemical or radiation therapy through the adaptation to intrinsic oxidative stress which confers drug resistance. This paper is focused on recent advances in the studies of the relation between oxidative stress, lipid peroxidation products, and cancer progression with particular attention to the pro-oxidant anticancer agents and the drug-resistant mechanisms, which could be modulated to ob |